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Research

Sodium selenate — living evidence map

The Atlas page holds the claims; this is how its evidence evolved.

Mechanism

In two tau-transgenic mouse strains, oral sodium selenate reduced tau hyperphosphorylation, prevented neurofibrillary tangles and neurodegeneration and improved memory and motor performance, acting through the phosphatase PP2A. · pmid:20643941

Identity

PubChem CID 25960: Sodium Selenate, Na2O4Se, 188.95 g/mol. · pubchem:25960

CAS registry number 13410-01-0. · pubchem:25960

IUPAC name: disodium;selenate. · pubchem:25960

Where the evidence actually sits

Preclinical only100%

Proportion of the 1 claim about Sodium selenate standing at each tier. Community volume is shown on the Consensus tab beside this and is never averaged into it.

Living evidence history

Sodium selenate mitigates tau pathology, neurodegeneration, and functional deficits in Alzheimer's disease models. (2010) · preclinical20102010

1 resolved citation, oldest to newest, coloured by tier. Retractions and contradictions land here as they are recorded.

Structure

No structure representation on record. PepJunkie draws only what an authoritative record supplies; for a peptide that is the residue sequence, and none has been curated for Sodium selenate yet.

Dose evidence, rung by rung

No established safe human dose. Nothing on file establishes one. That is a statement about the evidence PepJunkie holds, not a promise about the molecule.

  1. Approved-label dose · a resolved regulatory document names a labelled dose

    nothing resolved on this rung

  2. Studied human doses · resolved human studies at the established tier

    nothing resolved on this rung

  3. Observational evidence · resolved human data at the emerging tier: thin, or contested

    nothing resolved on this rung

  4. Community-reported protocols · protocol propositions from distinct members, counted once per human

    nothing resolved on this rung

  5. Preclinical evidence · resolved animal, cell or mechanistic work
    • Sodium selenate mitigates tau pathology, neurodegeneration, and functional deficits in Alzheimer's disease models. · pmid 20643941 · 2010

1 resolved citation, 0 unresolved (an unresolved citation lends nothing). Community volume is its own rung and is never averaged into the evidence rungs.